Abstract ‘Exhaustion’ is a term used to describe a state of native and redirected T-cell hypo-responsiveness resulting from persistent antigen exposure during chronic viral infections or cancer. Although a well-established phenotype across mice and humans, exhaustion at the molecular level remains poorly defined and inconsistent across the literature. This is, in part, due to an…
Oxford Open Immunology Template
Write in a clean editor, then format for Oxford Open Immunology in one click — DocuGuru applies the official Oxford University Press template with author–year references and exports a submission-ready PDF plus the editable LaTeX source. Free to start.
About the Oxford Open Immunology format
Oxford Open Immunology is a peer-reviewed journal published by Oxford University Press, covering SARS-CoV-2 and COVID-19 Research, COVID-19 Clinical Research Studies, T-cell and B-cell Immunology.
| Publisher | Oxford University Press |
|---|---|
| Reference style | Author–year (OUP) Author–year — (Smith, 2023) in the text Smith, A., Jones, B. and Lee, C. (2023) 'A representative article title', Oxford Open Immunology, 12(3), pp. 45–58.
Formats any DOI in the closest standard style — Oxford Open Immunology has no published style definition, so this is an approximation. No sign-up. |
| Publishes research in | SARS-CoV-2 and COVID-19 Research COVID-19 Clinical Research Studies T-cell and B-cell Immunology Immune Cell Function and Interaction Long-Term Effects of COVID-19 |
| ISSN | 2633-6960 |
| Citation impact (2-yr) | 4.51 |
| h-index | 21 |
| i10-index | 38 |
| Total citations | 1,133 |
| Article processing charge | $2,176 |
| Open access | Yes |
| Top institutions publishing here | University of Oxford |
| Journal website | academic.oup.com |
| You get | A submission-ready PDF and the editable LaTeX source — ready to submit. |
Papers published in Oxford Open Immunology per year
Citation impact of Oxford Open Immunology by publication year
Citations each year’s papers have accumulated so far — the most recent years are still building up.
Most-cited papers in Oxford Open Immunology
Severe Acute Respiratory Syndrome-Coronavirus-2 (SARS-CoV-2), the etiological agent of Coronavirus Diseases 2019 (COVID-19), triggers an adaptive immunity in the infected host that results in the production of virus-specific antibodies and T cells. Although kinetic and quantitative aspects of antibodies have been analyzed in large patient cohorts, similar information about SARS-CoV-2-specific T cells are scarce. We…
Orthostatic intolerance and other autonomic dysfunction syndromes are emerging as distinct symptom clusters in Long Covid. Often accompanying these are common, multi-system constitutional features such as fatigue, malaise and skin rashes which can signify generalized immune dysregulation. At the same time, multiple autoantibodies are identified in both Covid-related autonomic disorders and non-Covid autonomic disorders, implying…
SARS-CoV-2, the agent of the COVID-19 pandemic, emerged in late 2019 in China, and rapidly spread throughout the world to reach all continents. As the virus expanded in its novel human host, viral lineages diversified through the accumulation of around two mutations a month on average. Different viral lineages have replaced each other since the…
Abstract Infection with severe acute respiratory syndrom coronavirus 2 (SARS-CoV-2) is more likely to lead to poor outcomes in the elderly and those with cardiovascular disease, obesity or metabolic syndrome. Here, we consider mechanisms by which dyslipidaemia and the use of cholesterol-modifying drugs could influence the virus–host relationship. Cholesterol is essential for the assembly, replication…