Pregnancy outcomes in patients with pulmonary hypertension remain poor despite advanced therapies. Although consensus guidelines recommend against pregnancy in pulmonary hypertension, it may nonetheless occasionally occur. This guideline document sought to discuss the state of knowledge of pregnancy effects on pulmonary vascular disease and to define usual practice in avoidance of pregnancy and pregnancy management.…
Pulmonary Circulation Template
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About the Pulmonary Circulation format
Pulmonary Circulation is a peer-reviewed journal published by SAGE, covering Pulmonary Hypertension Research and Treatments, Cardiovascular Function and Risk Factors, Cardiovascular Issues in Pregnancy.
| Publisher | SAGE |
|---|---|
| Reference style | Author–year (Harvard) Author–year — (Smith, 2023) in the text Smith, A., Jones, B. and Lee, C. (2023) 'A representative article title', Pulmonary Circulation, 12(3), pp. 45–58.
Formats any DOI in Pulmonary Circulation style. No sign-up. |
| Publishes research in | Pulmonary Hypertension Research and Treatments Cardiovascular Function and Risk Factors Cardiovascular Issues in Pregnancy Heart Failure Treatment and Management Vascular Anomalies and Treatments |
| ISSN | 2045-8932 |
| Citation impact (2-yr) | 1.78 |
| h-index | 74 |
| i10-index | 909 |
| Total citations | 33,055 |
| Article processing charge | $2,200 |
| Open access | Yes |
| Top institutions publishing here | University of Colorado Denver |
| Journal website | pvri.info |
| You get | A submission-ready PDF and the editable LaTeX source — ready to submit. |
Papers published in Pulmonary Circulation per year
Citation impact of Pulmonary Circulation by publication year
Citations each year’s papers have accumulated so far — the most recent years are still building up.
Most-cited papers in Pulmonary Circulation
The endothelial monolayer partitioning underlying tissue from blood components in the vessel wall maintains tissue fluid balance and host defense through dynamically opening intercellular junctions. Edemagenic agonists disrupt endothelial barrier function by signaling the opening of the intercellular junctions leading to the formation of protein-rich edema in the interstitial tissue, a hallmark of tissue inflammation…
Current classifications of pulmonary hypertension have contributed a great deal to our understanding of pulmonary vascular disease, facilitated drug trials, and improved our understanding of congenital heart disease in adult survivors. However, these classifications are not applicable readily to pediatric disease. The classification system that we propose is based firmly in clinical practice. The specific…
Clinical trials have failed to demonstrate an effective preventative or therapeutic strategy for bronchopulmonary dysplasia (BPD), a multifactorial chronic lung disease in preterm infants frequently complicated by pulmonary hypertension (PH). Mesenchymal stem cells (MSCs) and their secreted components have been shown to prevent BPD and pulmonary fibrosis in rodent models. We hypothesized that treatment with…
Pulmonary hypertension (PH) is associated with structural and mechanical changes in the pulmonary vascular bed that increase right ventricular (RV) afterload. These changes, characterized by narrowing and stiffening, occur in both proximal and distal pulmonary arteries (PAs). An important consequence of arterial narrowing is increased pulmonary vascular resistance (PVR). Arterial stiffening, which can occur in…