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Skeletal Muscle is a peer-reviewed journal published by BMC (BioMed Central), covering Muscle Physiology and Disorders, Cardiomyopathy and Myosin Studies, Adipose Tissue and Metabolism. Over its lifetime it has published 536 papers, which have received 22,293 citations.
| Publisher | BMC (BioMed Central) |
|---|---|
| Reference style | Numbered (Vancouver) Numbered — [1], [2] in the text 1. Smith A, Jones B, Lee C. A representative article title. Skeletal Muscle. 2023;12(3):45–58.
Formats any DOI in Skeletal Muscle style. No sign-up. |
| Publishes research in | Muscle Physiology and Disorders Cardiomyopathy and Myosin Studies Adipose Tissue and Metabolism Neurogenetic and Muscular Disorders Research Mesenchymal stem cell research |
| ISSN | 2044-5040 |
| Citation impact (2-yr) | 4.06 |
| h-index | 75 |
| i10-index | 373 |
| Article processing charge | $2,195 |
| Open access | Yes — indexed in DOAJ |
| Top institutions publishing here | Inserm |
| Journal website | skeletalmusclejournal.biomedcentral.com |
| You get | A submission-ready PDF and the editable LaTeX source — ready to submit. |
Citations each year’s papers have accumulated so far — the most recent years are still building up.
The repair process of damaged tissue involves the coordinated activities of several cell types in response to local and systemic signals. Following acute tissue injury, infiltrating inflammatory cells and resident stem cells orchestrate their activities to restore tissue homeostasis. However, during chronic tissue damage, such as in muscular dystrophies, the inflammatory-cell infiltration and fibroblast activation…
A highly conserved signaling pathway involving insulin-like growth factor 1 (IGF1), and a cascade of intracellular components that mediate its effects, plays a major role in the regulation of skeletal muscle growth. A central component in this cascade is the kinase Akt, also called protein kinase B (PKB), which controls both protein synthesis, via the…
Developing skeletal muscles express unique myosin isoforms, including embryonic and neonatal myosin heavy chains, coded by the myosin heavy chain 3 (MYH3) and MYH8 genes, respectively, and myosin light chain 1 embryonic/atrial, encoded by the myosin light chain 4 (MYL4) gene. These myosin isoforms are transiently expressed during embryonic and fetal development and disappear shortly…
BACKGROUND: Sarcopenia, the loss of skeletal muscle mass during aging, increases the risk for falls and dependency. Resistance exercise (RE) training is an effective treatment to improve muscle mass and strength in older adults, but aging is associated with a smaller amount of training-induced hypertrophy. This may be due in part to an inability to…
BACKGROUND: Investigations into both the pathophysiology and therapeutic targets in muscle dystrophies have been hampered by the limited proliferative capacity of human myoblasts. Isolation of reliable and stable immortalized cell lines from patient biopsies is a powerful tool for investigating pathological mechanisms, including those associated with muscle aging, and for developing innovative gene-based, cell-based or…
