Reactive oxygen species (ROS) are generated as by-products of cellular metabolism, primarily in the mitochondria. When cellular production of ROS overwhelms its antioxidant capacity, damage to cellular macromolecules such as lipids, protein, and DNA may ensue. Such a state of "oxidative stress" is thought to contribute to the pathogenesis of a number of human diseases…
American Journal of Physiology-Lung Cellular and Molecular Physiology Template
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About the American Journal of Physiology-Lung Cellular and Molecular Physiology format
American Journal of Physiology-Lung Cellular and Molecular Physiology is a peer-reviewed journal published by American Physical Society, covering Neonatal Respiratory Health Research, Asthma and respiratory diseases, Neuroscience of respiration and sleep.
| Publisher | American Physical Society |
|---|---|
| Reference style | Numbered (APS) Numbered — [1], [2] in the text [1] A. Smith, B. Jones, and C. Lee, American Journal of Physiology-Lung Cellular and Molecular Physiology 12, 45 (2023).
Formats any DOI in American Journal of Physiology-Lung Cellular and Molecular Physiology style. No sign-up. |
| Publishes research in | Neonatal Respiratory Health Research Asthma and respiratory diseases Neuroscience of respiration and sleep Respiratory Support and Mechanisms Nitric Oxide and Endothelin Effects |
| ISSN | 1040-0605 |
| Citation impact (2-yr) | 3.38 |
| h-index | 209 |
| i10-index | 7,193 |
| Total citations | 436,237 |
| Top institutions publishing here | University of Colorado Denver |
| You get | A submission-ready PDF and the editable LaTeX source — ready to submit. |
Papers published in American Journal of Physiology-Lung Cellular and Molecular Physiology per year
Citation impact of American Journal of Physiology-Lung Cellular and Molecular Physiology by publication year
Citations each year’s papers have accumulated so far — the most recent years are still building up.
Most-cited papers in American Journal of Physiology-Lung Cellular and Molecular Physiology
Acute lung injury in humans is characterized histopathologically by neutrophilic alveolitis, injury of the alveolar epithelium and endothelium, hyaline membrane formation, and microvascular thrombi. Different animal models of experimental lung injury have been used to investigate mechanisms of lung injury. Most are based on reproducing in animals known risk factors for ARDS, such as sepsis,…
Nitric oxide and superoxide, which are produced by several cell types, rapidly combine to form peroxynitrite. This reaction can result in nitric oxide scavenging, and thus mitigation of the biological effects of superoxide. Also, superoxide can trap and hence modulate the effects of nitric oxide; superoxide dismutase, by controlling superoxide levels, therefore can influence the…
Single-walled carbon nanotubes (SWCNT) are new materials of emerging technological importance. As SWCNT are introduced into the life cycle of commercial products, their effects on human health and environment should be addressed. We demonstrated that pharyngeal aspiration of SWCNT elicited unusual pulmonary effects in C57BL/6 mice that combined a robust but acute inflammation with early…
Epithelial-mesenchymal transition (EMT), a process whereby fully differentiated epithelial cells undergo transition to a mesenchymal phenotype giving rise to fibroblasts and myofibroblasts, is increasingly recognized as playing an important role in repair and scar formation following epithelial injury. The extent to which this process contributes to fibrosis following injury in the lung is a subject…