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Frontiers in Molecular Neuroscience is a peer-reviewed journal published by Frontiers, covering Neuroscience and Neuropharmacology Research, Neuroinflammation and Neurodegeneration Mechanisms, Alzheimer's disease research and treatments. Over its lifetime it has published 4,494 papers, which have received 144,987 citations.
| Publisher | Frontiers |
|---|---|
| Reference style | Author–year (Harvard) Author–year — (Smith, 2023) in the text Smith, A., Jones, B. and Lee, C. (2023) 'A representative article title', Frontiers in Molecular Neuroscience, 12(3), pp. 45–58.
Formats any DOI in Frontiers in Molecular Neuroscience style. No sign-up. |
| Publishes research in | Neuroscience and Neuropharmacology Research Neuroinflammation and Neurodegeneration Mechanisms Alzheimer's disease research and treatments Neurogenesis and neuroplasticity mechanisms Genetics and Neurodevelopmental Disorders |
| ISSN | 1662-5099 |
| Citation impact (2-yr) | 4.73 |
| h-index | 144 |
| i10-index | 2,984 |
| Article processing charge | $2,950 |
| Open access | Yes — indexed in DOAJ |
| Top institutions publishing here | Inserm |
| Journal website | journal.frontiersin.org |
| You get | A submission-ready PDF and the editable LaTeX source — ready to submit. |
Citations each year’s papers have accumulated so far — the most recent years are still building up.
The phosphatidylinositol 3-kinase (PI3K)/AKT/mammalian target of rapamycin (mTOR) pathway is activated in the majority of human cancers. This pathway is known to play a key role in numerous cellular functions including proliferation, adhesion, migration, invasion, metabolism, and survival, but in the current review we focus on its role in angiogenesis. PI3K activation may occur via…
Rodents are commonly used to study the pathophysiological mechanisms of pain as studies in humans may be difficult to perform and ethically limited. As pain cannot be directly measured in rodents, many methods that quantify "pain-like" behaviors or nociception have been developed. These behavioral methods can be divided into stimulus-evoked or non-stimulus evoked (spontaneous) nociception,…
The NF-κB (nuclear factor κ-light-chain-enhancer of activated B cells) transcription factor family is a pleiotropic regulator of many cellular signaling pathways, providing a mechanism for the cells in response to a wide variety of stimuli linking to inflammation. The stimulated cells will be regulated by not only the canonical but also non-canonical NF-κB pathways. To…
TAR DNA binding protein 43 (TDP-43) is a versatile RNA/DNA binding protein involved in RNA-related metabolism. Hyper-phosphorylated and ubiquitinated TDP-43 deposits act as inclusion bodies in the brain and spinal cord of patients with the motor neuron diseases: amyotrophic lateral sclerosis (ALS) and frontotemporal lobar degeneration (FTLD). While the majority of ALS cases (90-95%) are…
Since its discovery almost three decades ago, the secreted neurotrophin brain-derived neurotrophic factor (BDNF) has been firmly implicated in the differentiation and survival of neurons of the CNS. More recently, BDNF has also emerged as an important regulator of synaptogenesis and synaptic plasticity mechanisms underlying learning and memory in the adult CNS. In this review…
