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Hereditary Cancer in Clinical Practice is a peer-reviewed journal published by BMC (BioMed Central), covering BRCA gene mutations in cancer, Genetic factors in colorectal cancer, Colorectal Cancer Screening and Detection. Over its lifetime it has published 842 papers, which have received 7,931 citations.
| Publisher | BMC (BioMed Central) |
|---|---|
| Reference style | Numbered (Vancouver) Numbered — [1], [2] in the text 1. Smith A, Jones B, Lee C. A representative article title. Hereditary Cancer in Clinical Practice. 2023;12(3):45–58.
Formats any DOI in Hereditary Cancer in Clinical Practice style. No sign-up. |
| Publishes research in | BRCA gene mutations in cancer Genetic factors in colorectal cancer Colorectal Cancer Screening and Detection Cancer Genomics and Diagnostics Genomics and Rare Diseases |
| ISSN | 1731-2302 |
| Citation impact (2-yr) | 2.49 |
| h-index | 41 |
| i10-index | 242 |
| Article processing charge | $2,195 |
| Open access | Yes — indexed in DOAJ |
| Top institutions publishing here | Pomeranian Medical University |
| Journal website | link.springer.com |
| You get | A submission-ready PDF and the editable LaTeX source — ready to submit. |
Citations each year’s papers have accumulated so far — the most recent years are still building up.
BACKGROUND: Cancer risk and Lhermitte-Duclos disease (LDD) risk estimates for Cowden syndrome (CS) are broad and based on a small number of patients. Risk estimates are vital to the development of diagnostic criteria, genetic counseling, and cancer surveillance. To further elaborate and estimate the risks associated with CS, a large cohort of patients was evaluated.…
Lynch syndrome (clinically referred to as HNPCC - Hereditary Non-Polyposis Colorectal Cancer) is a frequent, autosomal, dominantly-inherited cancer predisposition syndrome caused by various germline alterations that affect DNA mismatch repair genes, mainly MLH1 and MSH2. Patients inheriting this predisposition are susceptible to colorectal, endometrial and other extracolonic tumors. It has recently been shown that germline…
The association of germline mutations in the breast cancer susceptibility gene 1 (BRCA1) and the breast cancer susceptibility gene 2 (BRCA2) with the development of breast and ovarian cancers have been widely researched and recognised. It is known that these genes function at multiple sites in the body. Research has subsequently evolved into the connection…
The autosomal-dominant precancerous condition familial adenomatous polyposis (FAP) is caused by germline mutations in the tumour suppressor gene APC. Consistent correlations between the site of mutations in the gene and clinical phenotype have been published for different patient groups. We report our experiences of APC mutation analysis and genotype-phenotype correlations in 1166 unrelated polyposis families…
Colorectal cancer (CRC) is one of the most common forms of cancer worldwide and familial adenomatous polyposis (FAP) accounts for approximately 1% of all CRCs. Adenomatous polyposis syndromes can be divided into; familial adenomatous polyposis (FAP)classic FAP and attenuated familial adenomatous polyposis (AFAP), MUTYHassociated polyposis (MAP), NTHL1-associated polyposis (NAP) and polymerase proofreading-associated polyposis (PPAP). The…
