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Acta Neuropathologica Communications is a peer-reviewed journal published by BMC (BioMed Central), covering Alzheimer's disease research and treatments, Glioma Diagnosis and Treatment, Neuroinflammation and Neurodegeneration Mechanisms. Over its lifetime it has published 2,426 papers, which have received 95,233 citations.
| Publisher | BMC (BioMed Central) |
|---|---|
| Reference style | Numbered (Vancouver) Numbered — [1], [2] in the text 1. Smith A, Jones B, Lee C. A representative article title. Acta Neuropathologica Communications. 2023;12(3):45–58.
Formats any DOI in Acta Neuropathologica Communications style. No sign-up. |
| Publishes research in | Alzheimer's disease research and treatments Glioma Diagnosis and Treatment Neuroinflammation and Neurodegeneration Mechanisms Parkinson's Disease Mechanisms and Treatments Amyotrophic Lateral Sclerosis Research |
| ISSN | 2051-5960 |
| Citation impact (2-yr) | 4.72 |
| h-index | 130 |
| i10-index | 1,672 |
| Article processing charge | $2,931 |
| Open access | Yes — indexed in DOAJ |
| Top institutions publishing here | Inserm |
| Journal website | link.springer.com |
| You get | A submission-ready PDF and the editable LaTeX source — ready to submit. |
Citations each year’s papers have accumulated so far — the most recent years are still building up.
Alzheimer's disease (AD) is conceptualized as a progressive consequence of two hallmark pathological changes in grey matter: extracellular amyloid plaques and neurofibrillary tangles. However, over the past several years, neuroimaging studies have implicated micro- and macrostructural abnormalities in white matter in the risk and progression of AD, suggesting that in addition to the neuronal pathology…
INTRODUCTION: Microglia are tissue macrophages of the central nervous system that monitor brain homeostasis and react upon neuronal damage and stress. Aging and neurodegeneration induce a hypersensitive, pro-inflammatory phenotype, referred to as primed microglia. To determine the gene expression signature of priming, the transcriptomes of microglia in aging, Alzheimer's disease (AD), and amyotrophic lateral sclerosis…
The amyloid hypothesis has driven drug development strategies for Alzheimer's disease for over 20 years. We review why accumulation of amyloid-beta (Aβ) oligomers is generally considered causal for synaptic loss and neurodegeneration in AD. We elaborate on and update arguments for and against the amyloid hypothesis with new data and interpretations, and consider why the…
BACKGROUND: It is essential to determine the specificity of AV-1451 PET for tau in brain imaging by using pathological comparisons. We performed autoradiography in autopsy-confirmed Alzheimer disease and other neurodegenerative disorders to evaluate the specificity of AV-1451 binding for tau aggregates. METHODS: Tissue samples were selected that had a variety of dementia-related neuropathologies including Alzheimer…
Mutations in the PTEN-induced kinase 1 (PINK1) and Parkin RBR E3 ubiquitin-protein ligase (PARKIN) genes are associated with familial forms of Parkinson's disease (PD). PINK1, a protein kinase, and PARKIN, an E3 ubiquitin ligase, control the specific elimination of dysfunctional or superfluous mitochondria, thus fine-tuning mitochondrial network and preserving energy metabolism. PINK1 regulates PARKIN translocation…
