Write in a clean editor, then format for Cancer & Metabolism in one click — DocuGuru applies the official BMC (BioMed Central) template with numbered references and exports a submission-ready PDF plus the editable LaTeX source. Free to start.
Cancer & Metabolism is a peer-reviewed journal published by BMC (BioMed Central), covering Cancer, Hypoxia, and Metabolism, Metabolism, Diabetes, and Cancer, Cancer, Lipids, and Metabolism. Over its lifetime it has published 469 papers, which have received 17,156 citations.
| Publisher | BMC (BioMed Central) |
|---|---|
| Reference style | Numbered (Vancouver) Numbered — [1], [2] in the text 1. Smith A, Jones B, Lee C. A representative article title. Cancer & Metabolism. 2023;12(3):45–58.
Formats any DOI in Cancer & Metabolism style. No sign-up. |
| Publishes research in | Cancer, Hypoxia, and Metabolism Metabolism, Diabetes, and Cancer Cancer, Lipids, and Metabolism Metabolomics and Mass Spectrometry Studies Cancer Research and Treatments |
| ISSN | 2049-3002 |
| Citation impact (2-yr) | 5.06 |
| h-index | 68 |
| i10-index | 244 |
| Article processing charge | $2,440 |
| Open access | Yes — indexed in DOAJ |
| Top institutions publishing here | Cancer Research UK Scotland Institute |
| Journal website | link.springer.com |
| You get | A submission-ready PDF and the editable LaTeX source — ready to submit. |
Citations each year’s papers have accumulated so far — the most recent years are still building up.
Mitochondria produce reactive oxygen species (mROS) as a natural by-product of electron transport chain activity. While initial studies focused on the damaging effects of reactive oxygen species, a recent paradigm shift has shown that mROS can act as signaling molecules to activate pro-growth responses. Cancer cells have long been observed to have increased production of…
BACKGROUND: Metformin is widely used in the treatment of diabetes, and there is interest in 'repurposing' the drug for cancer prevention or treatment. However, the mechanism underlying the metabolic effects of metformin remains poorly understood. METHODS: We performed respirometry and stable isotope tracer analyses on cells and isolated mitochondria to investigate the impact of metformin…
Unchecked growth and proliferation is a hallmark of cancer, and numerous oncogenic mutations reprogram cellular metabolism to fuel these processes. As a central metabolic organelle, mitochondria execute critical biochemical functions for the synthesis of fundamental cellular components, including fatty acids, amino acids, and nucleotides. Despite the extensive interest in the glycolytic phenotype of many cancer…
BACKGROUND: Obesity is associated with increased recurrence and reduced survival of breast cancer. Adipocytes constitute a significant component of breast tissue, yet their role in provisioning metabolic substrates to support breast cancer progression is poorly understood. RESULTS: Here, we show that co-culture of breast cancer cells with adipocytes revealed cancer cell-stimulated depletion of adipocyte triacylglycerol.…
Both tumor hypoxia and dysregulated metabolism are classical features of cancer. Recent analyses have revealed complex interconnections between oncogenic activation, hypoxia signaling systems and metabolic pathways that are dysregulated in cancer. These studies have demonstrated that rather than responding simply to error signals arising from energy depletion or tumor hypoxia, metabolic and hypoxia signaling pathways…
