AIM: To explore changes in body weight and cardiometabolic risk factors after treatment withdrawal in the STEP 1 trial extension. MATERIALS AND METHODS: with ≥ 1 weight-related co-morbidity) without diabetes to 68 weeks of once-weekly subcutaneous semaglutide 2.4 mg (including 16 weeks of dose escalation) or placebo, as an adjunct to lifestyle intervention. At week…
Diabetes Obesity and Metabolism Template
Write in a clean editor, then format for Diabetes Obesity and Metabolism in one click — DocuGuru applies the official Wiley template with author–year references and exports a submission-ready PDF plus the editable LaTeX source. Free to start.
About the Diabetes Obesity and Metabolism format
Diabetes Obesity and Metabolism is a peer-reviewed journal published by Wiley, covering Diabetes Treatment and Management, Diabetes Management and Research, Diabetes, Cardiovascular Risks, and Lipoproteins.
| Publisher | Wiley |
|---|---|
| Reference style | Author–year (Chicago) Author–year — (Smith, 2023) in the text Smith, Ada, Ben Jones, and Cara Lee. 2023. "A Representative Article Title." Diabetes Obesity and Metabolism 12 (3): 45–58.
Formats any DOI in Diabetes Obesity and Metabolism style. No sign-up. |
| Publishes research in | Diabetes Treatment and Management Diabetes Management and Research Diabetes, Cardiovascular Risks, and Lipoproteins Pancreatic function and diabetes Metabolism, Diabetes, and Cancer |
| ISSN | 1462-8902 |
| Citation impact (2-yr) | 4.78 |
| h-index | 185 |
| i10-index | 4,088 |
| Total citations | 239,173 |
| Article processing charge | $4,940 |
| Top institutions publishing here | Novo Nordisk (Denmark) |
| Journal website | onlinelibrary.wiley.com |
| You get | A submission-ready PDF and the editable LaTeX source — ready to submit. |
Papers published in Diabetes Obesity and Metabolism per year
Citation impact of Diabetes Obesity and Metabolism by publication year
Citations each year’s papers have accumulated so far — the most recent years are still building up.
Most-cited papers in Diabetes Obesity and Metabolism
Incretin hormones are gut peptides that are secreted after nutrient intake and stimulate insulin secretion together with hyperglycaemia. GIP (glucose-dependent insulinotropic polypeptide) und GLP-1 (glucagon-like peptide-1) are the known incretin hormones from the upper (GIP, K cells) and lower (GLP-1, L cells) gut. Together, they are responsible for the incretin effect: a two- to three-fold…
AIMS: Sodium-glucose co-transporter 2 (SGLT2) reabsorbs glucose and sodium in the renal proximal tubule. Dapagliflozin, an SGLT2 inhibitor, targets hyperglycaemia in type 2 diabetes by increasing renal glucose excretion. To investigate whether the parallel occurring sodium loss would have diuretic-like physiologic effects, we compared dapagliflozin and hydrochlorothiazide (HCTZ) effects on 24-h blood pressure (BP), body…
Diabetic nephropathy (DN) is a major healthcare challenge. It occurs in up to 50% of those living with diabetes, is a major cause of end-stage kidney disease (ESKD) that requires treatment with dialysis or renal transplantation, and is associated with significantly increased cardiovascular morbidity and mortality. DN is a clinical syndrome characterized by persistent albuminuria…
Decreases in both beta-cell function and number can contribute to insulin deficiency in type 2 diabetes. Here, we quantified the beta-cell mass in pancreas obtained at autopsy of 57 type 2 diabetic (T2D) and 52 non-diabetic subjects of European origin. Sections from the body and tail were immunostained for insulin. The beta-cell mass was calculated…