Write in a clean editor, then format for Frontiers in Aging in one click — DocuGuru applies the official Frontiers template with author–year references and exports a submission-ready PDF plus the editable LaTeX source. Free to start.
Frontiers in Aging is a peer-reviewed journal published by Frontiers, covering Genetics, Aging, and Longevity in Model Organisms, Nutrition and Health in Aging, Frailty in Older Adults. Over its lifetime it has published 810 papers, which have received 7,667 citations.
| Publisher | Frontiers |
|---|---|
| Reference style | Author–year (Harvard) Author–year — (Smith, 2023) in the text Smith, A., Jones, B. and Lee, C. (2023) 'A representative article title', Frontiers in Aging, 12(3), pp. 45–58.
Formats any DOI in Frontiers in Aging style. No sign-up. |
| Publishes research in | Genetics, Aging, and Longevity in Model Organisms Nutrition and Health in Aging Frailty in Older Adults Telomeres, Telomerase, and Senescence Balance, Gait, and Falls Prevention |
| ISSN | 2673-6217 |
| Citation impact (2-yr) | 3.6 |
| h-index | 41 |
| i10-index | 238 |
| Article processing charge | $1,900 |
| Open access | Yes — indexed in DOAJ |
| Top institutions publishing here | Harvard University |
| Journal website | www.frontiersin.org |
| You get | A submission-ready PDF and the editable LaTeX source — ready to submit. |
Citations each year’s papers have accumulated so far — the most recent years are still building up.
With aging, there is increased dysfunction of both innate and adaptive immune responses, which contributes to impaired immune responses to pathogens and greater mortality and morbidity. This age-related immune dysfunction is defined in general as immunosenescence and includes an increase in the number of memory T cells, loss of ability to respond to antigen and…
). These mice have a shortened lifespan, stunted growth, renal disease, hyperphosphatemia, hypercalcemia, vascular calcification, cardiac hypertrophy, hypertension, pulmonary disease, cognitive impairment, multi-organ atrophy and fibrosis. Overexpression of Klotho has opposite effects, extending lifespan. In humans, Klotho levels decline with age, chronic kidney disease, diabetes, Alzheimer's disease and other conditions. Low Klotho levels correlate with…
The mechanistic Target of Rapamycin (mTOR) is a growth-related kinase that, in the context of the mTOR complex 1 (mTORC1), touches upon most fundamental cellular processes. Consequently, its activity is a critical determinant for cellular and organismal physiology, while its dysregulation is commonly linked to human aging and age-related disease. Presumably the most important stimulus…
The influence of the activation of a cellular phenotype termed senescence and it's importance in ageing and age-related diseases is becoming more and more evident. In fact, there is a huge effort to tackle these diseases via therapeutic drugs targeting senescent cells named senolytics. However, a clearer understanding of how senescence is activated and the…
