Write in a clean editor, then format for Journal of Neuroinflammation in one click — DocuGuru applies the official BMC (BioMed Central) template with numbered references and exports a submission-ready PDF plus the editable LaTeX source. Free to start.
Journal of Neuroinflammation is a peer-reviewed journal published by BMC (BioMed Central), covering Neuroinflammation and Neurodegeneration Mechanisms, Tryptophan and brain disorders, Multiple Sclerosis Research Studies. Over its lifetime it has published 4,913 papers, which have received 310,822 citations.
| Publisher | BMC (BioMed Central) |
|---|---|
| Reference style | Numbered (Vancouver) Numbered — [1], [2] in the text 1. Smith A, Jones B, Lee C. A representative article title. Journal of Neuroinflammation. 2023;12(3):45–58.
Formats any DOI in Journal of Neuroinflammation style. No sign-up. |
| Publishes research in | Neuroinflammation and Neurodegeneration Mechanisms Tryptophan and brain disorders Multiple Sclerosis Research Studies Neurogenesis and neuroplasticity mechanisms Alzheimer's disease research and treatments |
| ISSN | 1742-2094 |
| Citation impact (2-yr) | 9.67 |
| h-index | 216 |
| i10-index | 4,045 |
| Article processing charge | $2,931 |
| Open access | Yes — indexed in DOAJ |
| Top institutions publishing here | Inserm |
| Journal website | www.jneuroinflammation.com |
| You get | A submission-ready PDF and the editable LaTeX source — ready to submit. |
Citations each year’s papers have accumulated so far — the most recent years are still building up.
The concept of multiple macrophage activation states is not new. However, extending this idea to resident tissue macrophages, like microglia, has gained increased interest in recent years. Unfortunately, the research on peripheral macrophage polarization does not necessarily translate accurately to their central nervous system (CNS) counterparts. Even though pro- and anti-inflammatory cytokines can polarize microglia…
Stroke, the third leading cause of death and disability worldwide, is undergoing a change in perspective with the emergence of new ideas on neurodegeneration. The concept that stroke is a disorder solely of blood vessels has been expanded to include the effects of a detrimental interaction between glia, neurons, vascular cells, and matrix components, which…
Microglia make up the innate immune system of the central nervous system and are key cellular mediators of neuroinflammatory processes. Their role in central nervous system diseases, including infections, is discussed in terms of a participation in both acute and chronic neuroinflammatory responses. Specific reference is made also to their involvement in Alzheimer's disease where…
In this review, we first provide a brief historical perspective, discussing how peripheral nerve injury (PNI) may have caused World War I. We then consider the initiation, progression, and resolution of the cellular inflammatory response after PNI, before comparing the PNI inflammatory response with that induced by spinal cord injury (SCI).In contrast with central nervous…
BACKGROUND: A subset of patients with neuromyelitis optica spectrum disorders (NMOSD) has been shown to be seropositive for myelin oligodendrocyte glycoprotein antibodies (MOG-IgG). OBJECTIVE: To describe the epidemiological, clinical, radiological, cerebrospinal fluid (CSF), and electrophysiological features of a large cohort of MOG-IgG-positive patients with optic neuritis (ON) and/or myelitis (n = 50) as well as…
