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Science Signaling is a peer-reviewed journal published by AAAS (Science), covering Immune Cell Function and Interaction, Receptor Mechanisms and Signaling, T-cell and B-cell Immunology. Over its lifetime it has published 9,462 papers, which have received 225,517 citations.
| Publisher | AAAS (Science) |
|---|---|
| Reference style | Superscript numbered (Science) Superscript — small raised numerals in the text 1. A. Smith, B. Jones, C. Lee, A representative article title. Science Signaling 12, 45–58 (2023).
Formats any DOI in Science Signaling style. No sign-up. |
| Publishes research in | Immune Cell Function and Interaction Receptor Mechanisms and Signaling T-cell and B-cell Immunology Neuroscience and Neuropharmacology Research Protein Kinase Regulation and GTPase Signaling |
| ISSN | 1937-9145 |
| Citation impact (2-yr) | 5.36 |
| h-index | 195 |
| i10-index | 2,434 |
| Top institutions publishing here | American Association For The Advancement of Science |
| Journal website | stke.sciencemag.org |
| You get | A submission-ready PDF and the editable LaTeX source — ready to submit. |
Citations each year’s papers have accumulated so far — the most recent years are still building up.
The cBioPortal for Cancer Genomics (http://cbioportal.org) provides a Web resource for exploring, visualizing, and analyzing multidimensional cancer genomics data. The portal reduces molecular profiling data from cancer tissues and cell lines into readily understandable genetic, epigenetic, gene expression, and proteomic events. The query interface combined with customized data storage enables researchers to interactively explore genetic…
The epithelial-mesenchymal transition (EMT) is an essential mechanism in embryonic development and tissue repair. EMT also contributes to the progression of disease, including organ fibrosis and cancer. EMT, as well as a similar transition occurring in vascular endothelial cells called endothelial-mesenchymal transition (EndMT), results from the induction of transcription factors that alter gene expression to…
Eukaryotic cells replicate by a complex series of evolutionarily conserved events that are tightly regulated at defined stages of the cell division cycle. Progression through this cycle involves a large number of dedicated protein complexes and signaling pathways, and deregulation of this process is implicated in tumorigenesis. We applied high-resolution mass spectrometry-based proteomics to investigate…
Lysosomes are the major cellular site for clearance of defective organelles and digestion of internalized material. Demand on lysosomal capacity can vary greatly, and lysosomal function must be adjusted to maintain cellular homeostasis. Here, we identified an interaction between the lysosome-localized mechanistic target of rapamycin complex 1 (mTORC1) and the transcription factor TFEB (transcription factor…
Cytosolic double-stranded DNA (dsDNA) stimulates the production of type I interferon (IFN) through the endoplasmic reticulum (ER)-resident adaptor protein STING (stimulator of IFN genes), which activates the transcription factor interferon regulatory factor 3 (IRF3); however, how STING activates IRF3 is unclear. Here, we showed that STING stimulates phosphorylation of IRF3 by the kinase TBK1 (TANK-binding…
