Wiley

Glia Template

Write in a clean editor, then format for Glia in one click — DocuGuru applies the official Wiley template with author–year references and exports a submission-ready PDF plus the editable LaTeX source. Free to start.

About the Glia format

Glia is a peer-reviewed journal published by Wiley, covering Neuroinflammation and Neurodegeneration Mechanisms, Neurogenesis and neuroplasticity mechanisms, Neuroscience and Neuropharmacology Research.

PublisherWiley
Reference styleAuthor–year (Chicago)
Author–year — (Smith, 2023) in the text
Smith, Ada, Ben Jones, and Cara Lee. 2023. "A Representative Article Title." Glia 12 (3): 45–58.

Formats any DOI in Glia style. No sign-up.

Publishes research inNeuroinflammation and Neurodegeneration Mechanisms Neurogenesis and neuroplasticity mechanisms Neuroscience and Neuropharmacology Research Nerve injury and regeneration Ion channel regulation and function
ISSN0894-1491
Citation impact (2-yr)4.24
h-index234
i10-index4,670
Total citations376,995
Article processing charge$4,940
Top institutions publishing hereInserm
Journal websiteonlinelibrary.wiley.com
You getA submission-ready PDF and the editable LaTeX source — ready to submit.

Papers published in Glia per year

179
2014
188
2015
158
2016
211
2017
193
2018
227
2019
198
2020
191
2021
195
2022
180
2023
177
2024
181
2025

Citation impact of Glia by publication year

13.7K
2014
11.7K
2015
9.6K
2016
12.1K
2017
9.8K
2018
12K
2019
7.3K
2020
5K
2021
4.5K
2022
2.5K
2023
1.3K
2024
382
2025

Citations each year’s papers have accumulated so far — the most recent years are still building up.

Most-cited papers in Glia

Systemic LPS causes chronic neuroinflammation and progressive neurodegeneration

Liya Qin, Xuefei Wu, Michelle L. Block et al. · 3 Jan 2007

Inflammation is implicated in the progressive nature of neurodegenerative diseases, such as Parkinson's disease, but the mechanisms are poorly understood. A single systemic lipopolysaccharide (LPS, 5 mg/kg, i.p.) or tumor necrosis factor alpha (TNFalpha, 0.25 mg/kg, i.p.) injection was administered in adult wild-type mice and in mice lacking TNFalpha receptors (TNF R1/R2(-/-)) to discern the…

2,163 citations Cite SaveGo to paper →
Microglia as a source and target of cytokines

Uwe‐Karsten Hanisch · 4 Oct 2002

Cytokines constitute a significant portion of the immuno- and neuromodulatory messengers that can be released by activated microglia. By virtue of potent effects on resident and invading cells, microglial cyto- and chemokines regulate innate defense mechanisms, help the initiation and influence the type of immune responses, participate in the recruitment of leukocytes to the CNS,…

1,659 citations Cite SaveGo to paper →
Astrocyte activation and reactive gliosis

Milos Pekny, Michael Nilsson · 21 Apr 2005

Astrocytes become activated (reactive) in response to many CNS pathologies, such as stroke, trauma, growth of a tumor, or neurodegenerative disease. The process of astrocyte activation remains rather enigmatic and results in so-called "reactive gliosis," a reaction with specific structural and functional characteristics. In stroke or in CNS trauma, the lesion itself, the ischemic environment,…

1,629 citations Cite SaveGo to paper →
Immune function of astrocytes

Yuanshu Dong, Etty Benveniste · 2 Oct 2001

Astrocytes are the major glial cell within the central nervous system (CNS) and have a number of important physiological properties related to CNS homeostasis. The aspect of astrocyte biology addressed in this review article is the astrocyte as an immunocompetent cell within the brain. The capacity of astrocytes to express class II major histocompatibility complex…

1,291 citations Cite SaveGo to paper →
Immune function of microglia

Francesca Aloisi · 2 Oct 2001

During the past decade, mechanisms involved in the immune surveillance of the central nervous system (CNS) have moved to the forefront of neuropathological research mainly because of the recognition that most neurological disorders involve activation and, possibly, dysregulation of microglia, the intrinsic macrophages of the CNS. Increasing evidence indicates that, in addition to their well-established…

1,283 citations Cite SaveGo to paper →

Glia template — frequently asked questions

How do I write a paper in the Glia format?
In DocuGuru you write your manuscript in a normal editor — no LaTeX setup required — and select the Glia template. When you export, DocuGuru compiles the paper into the official Wiley format and hands you a submission-ready PDF along with the editable LaTeX source.
What reference style does Glia use?
Glia uses Author–year (Chicago) references, shown as author–year markers such as (Smith, 2023) in the text. DocuGuru formats every in-text citation and the reference list in this exact style automatically. A reference appears like this: Smith, Ada, Ben Jones, and Cara Lee. 2023. "A Representative Article Title." Glia 12 (3): 45–58.
Do I need to know LaTeX to submit to Glia?
No. DocuGuru generates the USG LaTeX class and compiles the PDF for you in the background, so you get a Wiley-ready Glia document without writing any LaTeX. If you do want it, the LaTeX source is included in the export.
Can I import an existing draft into the Glia template?
Yes. Paste or upload your current manuscript — Word, LaTeX, Markdown, or plain text — and DocuGuru reflows it into the Glia format with correct headings, figures, tables, and author–year citations.
Who publishes Glia?
Glia is a multidisciplinary journal published by Wiley. DocuGuru's Glia template matches Wiley's official submission format.
Can I export a submission-ready Glia PDF?
Yes — DocuGuru produces a PDF built with the official Glia template (the USG class) that is ready to submit to Wiley, together with the matching LaTeX source files.
How much does the Glia template cost?
You can start writing in the Glia template for free. Exporting the final submission-ready Glia PDF and LaTeX source is part of DocuGuru's paid plans — see the app for current pricing.
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