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Genome Instability & Disease is a peer-reviewed journal published by Springer Nature, covering DNA Repair Mechanisms, Epigenetics and DNA Methylation, CRISPR and Genetic Engineering.
| Publisher | Springer Nature |
|---|---|
| Reference style | Superscript numbered (Nature) Superscript — small raised numerals in the text 1. Smith, A., Jones, B. & Lee, C. A representative article title. Genome Instability & Disease 12, 45–58 (2023).
Formats any DOI in Genome Instability & Disease style. No sign-up. |
| Publishes research in | DNA Repair Mechanisms Epigenetics and DNA Methylation CRISPR and Genetic Engineering RNA modifications and cancer Cancer-related Molecular Pathways |
| ISSN | 2524-7662 |
| Citation impact (2-yr) | 2.05 |
| h-index | 15 |
| i10-index | 28 |
| Total citations | 1,112 |
| Article processing charge | $2,090 |
| Top institutions publishing here | Shenzhen University |
| Journal website | www.springer.com |
| You get | A submission-ready PDF and the editable LaTeX source — ready to submit. |
Citations each year’s papers have accumulated so far — the most recent years are still building up.
Ultraviolet (UV) irradiation causes various types of DNA damage, which leads to specific mutations and the emergence of skin cancer in humans, often decades after initial exposure. Different UV wavelengths cause the formation of prominent UV-induced DNA lesions. Most of these lesions are removed by the nucleotide excision repair pathway, which is defective in rare…
is the largest serine/threonine protein kinase of the phosphatidyl inositol 3-kinase-like protein kinase (PIKK) family and is the most highly expressed PIKK in human cells. With its DNA-binding partner Ku70/80, DNA-PKcs is required for regulated and efficient repair of ionizing radiation-induced DNA double-strand breaks via the non-homologous end joining (NHEJ) pathway. Loss of DNA-PKcs or…
Abstract N-acetyltransferase 10 catalyzes RNA N4-acetylcytidine (ac4C) modifications and thus regulates RNA stability and translation efficiency. However, the deacetylase for ac4C is unknown. SIRT7 was initially identified as an NAD + -dependent protein deacetylase and plays essential roles in genome stability, circadian rhythms, metabolism, and aging. In this study, we identified SIRT7 as a deacetylase…
Abstract A DNA double-strand break (DSB) is considered the most critical DNA lesion because it causes cell death and severe mutations if it is not repaired or repaired incorrectly. Accumulating evidence has shown that the majority of DSBs are repaired by DNA non-homologous end joining (NHEJ), the first utilized repair pathway in human cells. In…
