Fever is a hallmark symptom of disease across the animal kingdom. Yet, despite the evidence linking temperature fluctuation and immune response, much remains to be discovered about the molecular mechanisms governing these interactions. In patients with rheumatoid arthritis, for instance, it is clinically accepted that joint temperature can predict disease progression. But it was only…
Discovery Immunology Template
Write in a clean editor, then format for Discovery Immunology in one click — DocuGuru applies the official Oxford University Press template with author–year references and exports a submission-ready PDF plus the editable LaTeX source. Free to start.
About the Discovery Immunology format
Discovery Immunology is a peer-reviewed journal published by Oxford University Press, covering Immune Cell Function and Interaction, T-cell and B-cell Immunology, Immunotherapy and Immune Responses.
| Publisher | Oxford University Press |
|---|---|
| Reference style | Author–year (OUP) Author–year — (Smith, 2023) in the text Smith, A., Jones, B. and Lee, C. (2023) 'A representative article title', Discovery Immunology, 12(3), pp. 45–58.
Formats any DOI in the closest standard style — Discovery Immunology has no published style definition, so this is an approximation. No sign-up. |
| Publishes research in | Immune Cell Function and Interaction T-cell and B-cell Immunology Immunotherapy and Immune Responses IL-33, ST2, and ILC Pathways Immune Response and Inflammation |
| ISSN | 2754-2483 |
| Citation impact (2-yr) | 3.55 |
| h-index | 13 |
| i10-index | 21 |
| Total citations | 566 |
| Open access | Yes |
| Top institutions publishing here | University of Glasgow |
| You get | A submission-ready PDF and the editable LaTeX source — ready to submit. |
Papers published in Discovery Immunology per year
Citation impact of Discovery Immunology by publication year
Citations each year’s papers have accumulated so far — the most recent years are still building up.
Most-cited papers in Discovery Immunology
Monocytes are a key component of the innate immune system. They undergo intricate developmental processes within the bone marrow, leading to diverse monocyte subsets in the circulation. In a state of healthy homeostasis, monocytes are continuously released into the bloodstream, destined to repopulate specific tissue-resident macrophage pools where they fulfil tissue-specific functions. However, under pathological…
Abstract In lymphocytes, Nr4a gene expression is specifically regulated by antigen receptor signalling, making them ideal targets for use as distal T cell receptor (TCR) reporters. Nr4a3-Timer of cell kinetics and activity (Tocky) mice are a ground-breaking tool to report TCR-driven Nr4a3 expression using Fluorescent Timer protein (FT). FT undergoes a time-dependent shift in its…
Interleukin (IL)-33 is highly expressed in the nucleus of cells present at barrier sites and signals via the ST2 receptor. IL-33 signalling via ST2 is essential for return to tissue homeostasis after acute inflammation, promoting fibrinogenesis and wound healing at injury sites. However, this wound-healing response becomes aberrant during chronic or sustained inflammation, leading to…
There is an intriguing dichotomy in the function of cytokine interleukin-15-at low levels, it is required for the homeostasis of the immune system, yet when it is upregulated in response to pathogenic infections or in autoimmunity, IL-15 drives inflammation. IL-15 associates with the IL-15Rα within both myeloid and non-haematopoietic cells, where IL-15Rα trans-presents IL-15 in…