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Carcinogenesis is a peer-reviewed journal published by Oxford University Press, covering Carcinogens and Genotoxicity Assessment, DNA Repair Mechanisms, Genomics, phytochemicals, and oxidative stress. Over its lifetime it has published 14,238 papers, which have received 728,953 citations.
| Publisher | Oxford University Press |
|---|---|
| Reference style | Numbered (OUP) Numbered — [1], [2] in the text [1] A. Smith, B. Jones, and C. Lee, A representative article title, Carcinogenesis 12 (2023) 45–58.
Formats any DOI in the closest standard style — Carcinogenesis has no published style definition, so this is an approximation. No sign-up. |
| Publishes research in | Carcinogens and Genotoxicity Assessment DNA Repair Mechanisms Genomics, phytochemicals, and oxidative stress Epigenetics and DNA Methylation Cancer-related Molecular Pathways |
| ISSN | 0143-3334 |
| Citation impact (2-yr) | 2.69 |
| h-index | 262 |
| i10-index | 11,142 |
| Article processing charge | $4,428 |
| Top institutions publishing here | National Cancer Institute |
| Journal website | carcin.oxfordjournals.org |
| You get | A submission-ready PDF and the editable LaTeX source — ready to submit. |
Citations each year’s papers have accumulated so far — the most recent years are still building up.
Inflammatory conditions in selected organs increase the risk of cancer. An inflammatory component is present also in the microenvironment of tumors that are not epidemiologically related to inflammation. Recent studies have begun to unravel molecular pathways linking inflammation and cancer. In the tumor microenvironment, smoldering inflammation contributes to proliferation and survival of malignant cells, angiogenesis,…
Epigenetic mechanisms are essential for normal development and maintenance of tissue-specific gene expression patterns in mammals. Disruption of epigenetic processes can lead to altered gene function and malignant cellular transformation. Global changes in the epigenetic landscape are a hallmark of cancer. The initiation and progression of cancer, traditionally seen as a genetic disease, is now…
An antioxidant fraction of Chinese green tea (green tea antioxidant; GTA), containing several catechins, has been previously shown to inhibit 12-O-tetradecanoylphorbol-13-acetate (TPA)-induced tumor promotion in mouse skin. In the present study, GTA was shown to have antioxidative activity toward hydrogen peroxide (H2O2) and the superoxide radical (O2-). GTA also prevented oxygen radical and H2O2-induced cytotoxicity…
A major development of carcinogenesis research in the past 20 years has been the discovery of significant levels of DNA damage arising from endogenous cellular sources. Dramatic improvements in analytical chemistry have provided sensitive and specific methodology for identification and quantitation of DNA adducts. Application of these techniques to the analysis of nuclear DNA from…
In the last decade, basic cancer research has produced remarkable advances in our understanding of cancer biology and cancer genetics. Among the most important of these advances is the realization that apoptosis and the genes that control it have a profound effect on the malignant phenotype. For example, it is now clear that some oncogenic…
