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Research and Practice in Thrombosis and Haemostasis is a peer-reviewed journal published by Elsevier, covering Venous Thromboembolism Diagnosis and Management, Hemophilia Treatment and Research, Platelet Disorders and Treatments.
| Publisher | Elsevier |
|---|---|
| Reference style | Numbered (Elsevier) Numbered — [1], [2] in the text [1] A. Smith, B. Jones, C. Lee, A representative article title, Research and Practice in Thrombosis and Haemostasis 12 (2023) 45–58.
Formats any DOI in Research and Practice in Thrombosis and Haemostasis style. No sign-up. |
| Publishes research in | Venous Thromboembolism Diagnosis and Management Hemophilia Treatment and Research Platelet Disorders and Treatments Blood Coagulation and Thrombosis Mechanisms Heparin-Induced Thrombocytopenia and Thrombosis |
| ISSN | 2475-0379 |
| Citation impact (2-yr) | 1.79 |
| h-index | 56 |
| i10-index | 627 |
| Total citations | 22,074 |
| Article processing charge | $2,000 |
| Open access | Yes |
| Top institutions publishing here | Leiden University Medical Center |
| Journal website | www.journals.elsevier.com |
| You get | A submission-ready PDF and the editable LaTeX source — ready to submit. |
Citations each year’s papers have accumulated so far — the most recent years are still building up.
Since its discovery over 350 years ago, studies of fibrinogen have revealed remarkable characteristics. Its complex structure as a large (340 kDa) hexameric homodimer supports complex roles in hemostasis and homeostasis. Fibrinogen synthesis is regulated at the transcriptional and translational levels, undergoing both constitutive (basal) secretion from liver, and inducible upregulation in response to inflammatory…
. In the buildup of a platelet-fibrin thrombus, the extrinsic, tissue factor-driven coagulation pathway is predominant in early stages, while the intrinsic, factor XII pathway seems to promote at later time points. Already early generation of thrombin enforces platelet responses and stimulates intra-thrombus heterogeneity with patches of loosely aggregated, contracted, and phosphatidylserine-exposing platelets. Fibrin actively…
Blood coagulation is initiated by exposure of blood to the transmembrane protein tissue factor (TF). 1 Under normal conditions, TF is not expressed by cells, such as circulating blood cells and endothelial cells, which are in direct contact with plasma-circulating coagulation protease zymogens. 1,2 However, subendothelial cells including pericytes, fibroblasts, and smooth muscle cells constitutively…
Beyond their role in hemostasis and thrombosis, platelets are increasingly recognized as key regulators of the inflammatory response under sterile and infectious conditions. Both platelet receptors and secretion are critical for these functions and contribute to their interaction with the endothelium and innate immune system. Platelet-leukocyte interactions are increased in thrombo-inflammatory diseases and are sensitive…
Patients with liver diseases may develop alterations in all components of the hemostatic system. Thrombocytopenia, low levels of coagulation factors and inhibitors, low levels of fibrinolytic proteins, and increased levels of endothelial-derived proteins such as von Willebrand factor are all part of the coagulopathy of liver disease. Due to concomitant changes in pro- and antihemostatic…
