Autoimmune diseases cause significant and chronic morbidity and disability. The actual number of persons in the United States that are affected by autoimmune diseases and the resultant magnitude of their impact on the public's health are limited to a few specific diseases. In order to understand the clinical, public health and economic importance of these…
Clinical Immunology and Immunopathology Template
Write in a clean editor, then format for Clinical Immunology and Immunopathology in one click — DocuGuru applies the official Elsevier template with numbered references and exports a submission-ready PDF plus the editable LaTeX source. Free to start.
About the Clinical Immunology and Immunopathology format
Clinical Immunology and Immunopathology is a peer-reviewed journal published by Elsevier, covering T-cell and B-cell Immunology, Immune Cell Function and Interaction, Monoclonal and Polyclonal Antibodies Research.
| Publisher | Elsevier |
|---|---|
| Reference style | Numbered (Elsevier) Numbered — [1], [2] in the text [1] A. Smith, B. Jones, C. Lee, A representative article title, Clinical Immunology and Immunopathology 12 (2023) 45–58.
Formats any DOI in Clinical Immunology and Immunopathology style. No sign-up. |
| Publishes research in | T-cell and B-cell Immunology Immune Cell Function and Interaction Monoclonal and Polyclonal Antibodies Research Immunodeficiency and Autoimmune Disorders Systemic Lupus Erythematosus Research |
| ISSN | 0090-1229 |
| h-index | 123 |
| i10-index | 2,926 |
| Total citations | 128,099 |
| Top institutions publishing here | Medical University of South Carolina |
| You get | A submission-ready PDF and the editable LaTeX source — ready to submit. |
Papers published in Clinical Immunology and Immunopathology per year
Citation impact of Clinical Immunology and Immunopathology by publication year
Citations each year’s papers have accumulated so far — the most recent years are still building up.
Most-cited papers in Clinical Immunology and Immunopathology
The immunosuppressants cyclosporin A (CsA), FK506, and rapamycin suppress the immune response by inhibiting evolutionary conserved signal transduction pathways. CsA, FK506, and rapamycin bind to their intracellular receptors, immunophilins, creating composite surfaces that block the activity of specific targets. For CsA/cyclophilin and FK506/FKBP the target is calcineurin. Because of the large surface area of interaction…