The Cancer Genome Atlas revealed the genomic landscapes of human cancers. In parallel, immunotherapy is transforming the treatment of advanced cancers. Unfortunately, the majority of patients do not respond to immunotherapy, making the identification of predictive markers and the mechanisms of resistance an area of intense research. To increase our understanding of tumor-immune cell interactions,…
Cell Reports Template
Write in a clean editor, then format for Cell Reports in one click — DocuGuru applies the official Elsevier template with numbered references and exports a submission-ready PDF plus the editable LaTeX source. Free to start.
About the Cell Reports format
Cell Reports is a peer-reviewed journal published by Elsevier, covering RNA Research and Splicing, Neuroscience and Neuropharmacology Research, RNA modifications and cancer.
| Publisher | Elsevier |
|---|---|
| Reference style | Numbered (Elsevier) Numbered — [1], [2] in the text [1] A. Smith, B. Jones, C. Lee, A representative article title, Cell Reports 12 (2023) 45–58.
Formats any DOI in Cell Reports style. No sign-up. |
| Publishes research in | RNA Research and Splicing Neuroscience and Neuropharmacology Research RNA modifications and cancer Immune Cell Function and Interaction Epigenetics and DNA Methylation |
| ISSN | 2211-1247 |
| Citation impact (2-yr) | 7.87 |
| h-index | 325 |
| i10-index | 13,373 |
| Total citations | 1,051,057 |
| Article processing charge | $5,200 |
| Open access | Yes |
| Top institutions publishing here | Harvard University |
| Journal website | www.sciencedirect.com |
| You get | A submission-ready PDF and the editable LaTeX source — ready to submit. |
Papers published in Cell Reports per year
Citation impact of Cell Reports by publication year
Citations each year’s papers have accumulated so far — the most recent years are still building up.
Most-cited papers in Cell Reports
Topologically associating domains (TADs) are fundamental structural and functional building blocks of human interphase chromosomes, yet the mechanisms of TAD formation remain unclear. Here, we propose that loop extrusion underlies TAD formation. In this process, cis-acting loop-extruding factors, likely cohesins, form progressively larger loops but stall at TAD boundaries due to interactions with boundary proteins,…
PD-L1 and PD-L2 are ligands for the PD-1 immune inhibiting checkpoint that can be induced in tumors by interferon exposure, leading to immune evasion. This process is important for immunotherapy based on PD-1 blockade. We examined the specific molecules involved in interferon-induced signaling that regulates PD-L1 and PD-L2 expression in melanoma cells. These studies revealed…
We established a collection of 7,000 transgenic lines of Drosophila melanogaster. Expression of GAL4 in each line is controlled by a different, defined fragment of genomic DNA that serves as a transcriptional enhancer. We used confocal microscopy of dissected nervous systems to determine the expression patterns driven by each fragment in the adult brain and…
Spontaneous tumor-initiated T cell priming is dependent on IFN-β production by tumor-resident dendritic cells. On the basis of recent observations indicating that IFN-β expression was dependent upon activation of the host STING pathway, we hypothesized that direct engagement of STING through intratumoral (IT) administration of specific agonists would result in effective anti-tumor therapy. After proof-of-principle…