The NF-κB family of transcription factors has an essential role in inflammation and innate immunity. Furthermore, NF-κB is increasingly recognized as a crucial player in many steps of cancer initiation and progression. During these latter processes NF-κB cooperates with multiple other signaling molecules and pathways. Prominent nodes of crosstalk are mediated by other transcription factors…
Molecular Cancer Template
Write in a clean editor, then format for Molecular Cancer in one click — DocuGuru applies the official BMC (BioMed Central) template with numbered references and exports a submission-ready PDF plus the editable LaTeX source. Free to start.
About the Molecular Cancer format
Molecular Cancer is a peer-reviewed journal published by BMC (BioMed Central), covering Cancer-related molecular mechanisms research, MicroRNA in disease regulation, Circular RNAs in diseases. Over its lifetime it has published 3,991 papers, which have received 418,285 citations.
| Publisher | BMC (BioMed Central) |
|---|---|
| Reference style | Numbered (Vancouver) Numbered — [1], [2] in the text 1. Smith A, Jones B, Lee C. A representative article title. Molecular Cancer. 2023;12(3):45–58.
Formats any DOI in Molecular Cancer style. No sign-up. |
| Publishes research in | Cancer-related molecular mechanisms research MicroRNA in disease regulation Circular RNAs in diseases Cancer Cells and Metastasis RNA modifications and cancer |
| ISSN | 1476-4598 |
| Citation impact (2-yr) | 26.56 |
| h-index | 292 |
| i10-index | 3,349 |
| Article processing charge | $3,888 |
| Open access | Yes — indexed in DOAJ |
| Top institutions publishing here | Sun Yat-sen University |
| Journal website | molecular-cancer.biomedcentral.com |
| You get | A submission-ready PDF and the editable LaTeX source — ready to submit. |
Papers published in Molecular Cancer per year
Citation impact of Molecular Cancer by publication year
Citations each year’s papers have accumulated so far — the most recent years are still building up.
Most-cited papers in Molecular Cancer
Cancer-associated fibroblasts (CAFs), a stromal cell population with cell-of-origin, phenotypic and functional heterogeneity, are the most essential components of the tumor microenvironment (TME). Through multiple pathways, activated CAFs can promote tumor growth, angiogenesis, invasion and metastasis, along with extracellular matrix (ECM) remodeling and even chemoresistance. Numerous previous studies have confirmed the critical role of the…
AIM: Clinical resistance is a complex phenomenon in major human cancers involving multifactorial mechanisms, and hypoxia is one of the key components that affect the cellular expression program and lead to therapy resistance. The present study aimed to summarize the role of hypoxia in cancer therapy by regulating the tumor microenvironment (TME) and to highlight…
The PI3K/AKT/mTOR (PAM) signaling pathway is a highly conserved signal transduction network in eukaryotic cells that promotes cell survival, cell growth, and cell cycle progression. Growth factor signalling to transcription factors in the PAM axis is highly regulated by multiple cross-interactions with several other signaling pathways, and dysregulation of signal transduction can predispose to cancer…
BACKGROUND: Recently, a small population of cancer stem cells in adult and pediatric brain tumors has been identified. Some evidence has suggested that CD133 is a marker for a subset of leukemia and glioblastoma cancer stem cells. Especially, CD133 positive cells isolated from human glioblastoma may initiate tumors and represent novel targets for therapeutics. The…